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The main consequence of the dysregulation of this part of human metabolism is undoubtedly the development of atherosclerosis.
Unfortunately, the usual medical approach to these issues, except for particular exceptions, typically begins subtly around the age of 40 and more effectively around the age of 50.
Therefore, it should be a concern of healthcare professionals to provide appropriate follow-up for their patients from that age onwards, to prevent reaching points of difficult reversibility when patients are 60 or older.
The first two parts of this series on cholesterol discussed the need to clarify some notions about this topic that can help us better understand the impact of this molecule on the body, as well as to know the best dietary interventions to optimize our health in this area. With this final part, the aim is to integrate this information so that I can explain my perspective on the most appropriate way to approach this issue, in light of my interpretation of current knowledge.
However, cholesterol and triglycerides are the main "cargo" transported by lipoproteins, and among these, LDLs are the lipoproteins with the highest atherogenic potential. As we saw in the previous text, the higher the number of LDL particles, the greater the possibility of atherosclerosis development.
Thus, how can we shape our diet to decrease the number of LDL particles and not promote the emergence of atherosclerotic plaques in our vessels?
